{"id":75,"date":"2026-01-08T09:40:54","date_gmt":"2026-01-08T12:40:54","guid":{"rendered":"https:\/\/ingenieria.udd.cl\/doctorado\/?post_type=person&#038;p=75"},"modified":"2026-01-28T15:14:45","modified_gmt":"2026-01-28T18:14:45","slug":"juan-francisco-calderon","status":"publish","type":"person","link":"https:\/\/ingenieria.udd.cl\/doctorado-en-ingenieria-aplicada\/persona\/juan-francisco-calderon\/","featured_media":76,"template":"","people_tax":[],"people_areas":[],"people_levels":[],"class_list":["post-75","person","type-person","status-publish","has-post-thumbnail","hentry"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v26.9 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>Juan Francisco Calder\u00f3n - Doctorado en Ingenier\u00eda Aplicada<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/ingenieria.udd.cl\/doctorado-en-ingenieria-aplicada\/persona\/juan-francisco-calderon\/\" \/>\n<meta property=\"og:locale\" content=\"es_ES\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Juan Francisco Calder\u00f3n - 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Sus l\u00edneas de investigaci\u00f3n se centran en la variaci\u00f3n gen\u00e9tica y su efecto en fenotipos de inter\u00e9s cl\u00ednico.<\/p>\n<p>Intereses<\/p>\n<p>Su inter\u00e9s principal de investigaci\u00f3n es el uso de modelos animales para comprender el papel de la variaci\u00f3n gen\u00e9tica en la modulaci\u00f3n de fenotipos, especialmente en el sistema cardiovascular. En etapas previas de su carrera, centr\u00f3 su trabajo en la biolog\u00eda del desarrollo de cardiopat\u00edas cong\u00e9nitas, utilizando modelos murinos con mutaciones en genes que codifican elementos clave de la v\u00eda de se\u00f1alizaci\u00f3n TGF\u03b2, las que est\u00e1n asociadas a enfermedades mendelianas como el S\u00edndrome de Loeys-Dietz (LDS), entre otras. La identificaci\u00f3n de genes que interact\u00faan con esta v\u00eda abre nuevas posibilidades terap\u00e9uticas con potencial aplicaci\u00f3n cl\u00ednica.<\/p>\n<p>Asimismo, le interesan las t\u00e9cnicas de ingenier\u00eda gen\u00e9tica aplicadas tanto en l\u00edneas celulares como en la generaci\u00f3n de modelos animales que reproduzcan variaciones gen\u00e9ticas con posible efecto modulador de fenotipos. Este enfoque permite estudiar dichas variaciones en el contexto gen\u00f3mico \u201cnativo\u201d del organismo.<\/p>\n<p>Interests<\/p>\n<p>His main research interest is to understand the role of genetic variation in the modulation of phenotypes, particularly within the cardiovascular system, using animal models. In previous work, he focused on the biology of congenital heart defects in murine models carrying mutations in genes that code for key components of the TGF\u03b2 signaling pathway, which cause Mendelian disorders such as Loeys-Dietz Syndrome (LDS). Identifying elements that interact with this pathway provides new potential therapeutic targets for patients with these diseases.<\/p>\n<p>Another line of interest is the use of genome engineering techniques, both in cell culture and in the generation of mouse models that reflect elements of genetic variation involved in phenotype modification, enabling the study of their effects in the \u201cnative\u201d genetic context of the host.<\/p>\n<p>Publicaciones seleccionadas<\/p>\n<p>Small molecule inhibitor screening identified HSP90 inhibitor 17-AAG as potential therapeutic agent for gallbladder cancer. Weber H, Valbuena JR, Barbhuiya MA, Stein S, Kunkel H, Garc\u00eda P, Bizama C, Riquelme I, Espinoza JA, Kurtz SE, Tyner JW, Calderon JF, Corval\u00e1n AH, Grez M, Pandey A, Leal-Rojas P, Roa JC. Oncotarget. 2017. doi: 10.18632\/oncotarget.15410.<\/p>\n<p>Regulation of Connexins Expression Levels by MicroRNAs, an Update. Calder\u00f3n JF, Retamal MA. Front Physiol. 2016;7:558.<\/p>\n<p>Elevated expression levels of lysyl oxidases protect against aortic aneurysm progression in Marfan syndrome. Busnadiego O, del Blanco D, Gonzalez-Santamar\u00eda J, Habashi J, Calder\u00f3n JF, et al. Journal of Molecular and Cellular Cardiology. 2015.<\/p>\n<p>Current Controversies in Diagnosis and Management of Cleft Palate and Velopharyngeal Insufficiency. Ysunza PA, Repetto GM, Pamplona MC, Calder\u00f3n JF, et al. BioMed Research International. 2015; Article ID 196240.<\/p>\n<p>Angiotensin II-dependent TGF-\u03b2 signaling contributes to Loeys-Dietz syndrome vascular pathogenesis. Gallo EM, Loch DC, Habashi JP, Calderon JF, et al. J Clin Invest. 2014;124(1):448-60.<\/p>\n<p>GluA3-deficiency in mice is associated with increased social and aggressive behavior and elevated dopamine in striatum. Adamczyk A, Mejias R, Takamiya K, Yocum J, Krasnova I, Calder\u00f3n JF, Cadet JL, Huganir R, Pletnikov M, Wang T. Behavioural Brain Research. 2012;229(1):265-72.<\/p>\n<p>VEGFA polymorphisms and cardiovascular anomalies in 22q11 microdeletion syndrome. Calder\u00f3n JF, Puga AR, Guzm\u00e1n ML, Astete CP, et al. Biol Res. 2009;42(4):461-8.<\/p>\n<p>Clinical features of chromosome 22q11.2 microdeletion syndrome in 208 Chilean patients. Repetto GM, Guzm\u00e1n ML, Puga A, Calder\u00f3n JF, et al. Clin Genet. 2009;76(5):465-70.<\/p>\n<p>Reconocimientos<\/p>\n<p>ASHG\/Charles J. Epstein Trainee Award for Excellence in Human Genetics Research. American Society of Human Genetics, Boston, MA.<\/p>\n<p>Fulbright \u2013 CONICYT Predoctoral Training Scholarship. Santiago, Chile.<\/p>\n\t\t\t\t\t\t\t\t\t\t<p><strong>L\u00ednea de Investigaci\u00f3n:<\/strong><\/p>\n<p>Ingenier\u00eda de Sistemas de Salud y Biomedicina<\/p>\n<ul>\n<li><em>Uso de la inteligencia artificial para el an\u00e1lisis avanzado de datos biom\u00e9dicos.<\/em><\/li>\n<\/ul>\n\t\t<\/div>\n\t<\/div>\n<\/div>\n"},"identifier":"cc754f82d62b74580ed968e5d09a9a79a9ef0eb8","has_credential":[],"given_name":"Juan Francisco","family_name":"Calder\u00f3n","name":"Juan Francisco Calder\u00f3n","job_title":"Investigador docente","telephone":"","note":"Ingeniero en Biotecnolog\u00eda Molecular por la Universidad de Chile y Ph.D. en Gen\u00e9tica Humana y Biolog\u00eda Molecular por The Johns Hopkins University, Baltimore, MD (USA). Sus l\u00edneas de investigaci\u00f3n se centran en la variaci\u00f3n gen\u00e9tica y su efecto en fenotipos de inter\u00e9s cl\u00ednico.\r\n\r\nIntereses\r\n\r\nSu inter\u00e9s principal de investigaci\u00f3n es el uso de modelos animales para comprender el papel de la variaci\u00f3n gen\u00e9tica en la modulaci\u00f3n de fenotipos, especialmente en el sistema cardiovascular. En etapas previas de su carrera, centr\u00f3 su trabajo en la biolog\u00eda del desarrollo de cardiopat\u00edas cong\u00e9nitas, utilizando modelos murinos con mutaciones en genes que codifican elementos clave de la v\u00eda de se\u00f1alizaci\u00f3n TGF\u03b2, las que est\u00e1n asociadas a enfermedades mendelianas como el S\u00edndrome de Loeys-Dietz (LDS), entre otras. La identificaci\u00f3n de genes que interact\u00faan con esta v\u00eda abre nuevas posibilidades terap\u00e9uticas con potencial aplicaci\u00f3n cl\u00ednica.\r\n\r\nAsimismo, le interesan las t\u00e9cnicas de ingenier\u00eda gen\u00e9tica aplicadas tanto en l\u00edneas celulares como en la generaci\u00f3n de modelos animales que reproduzcan variaciones gen\u00e9ticas con posible efecto modulador de fenotipos. Este enfoque permite estudiar dichas variaciones en el contexto gen\u00f3mico \u201cnativo\u201d del organismo.\r\n\r\nInterests\r\n\r\nHis main research interest is to understand the role of genetic variation in the modulation of phenotypes, particularly within the cardiovascular system, using animal models. In previous work, he focused on the biology of congenital heart defects in murine models carrying mutations in genes that code for key components of the TGF\u03b2 signaling pathway, which cause Mendelian disorders such as Loeys-Dietz Syndrome (LDS). Identifying elements that interact with this pathway provides new potential therapeutic targets for patients with these diseases.\r\n\r\nAnother line of interest is the use of genome engineering techniques, both in cell culture and in the generation of mouse models that reflect elements of genetic variation involved in phenotype modification, enabling the study of their effects in the \u201cnative\u201d genetic context of the host.\r\n\r\nPublicaciones seleccionadas\r\n\r\nSmall molecule inhibitor screening identified HSP90 inhibitor 17-AAG as potential therapeutic agent for gallbladder cancer. Weber H, Valbuena JR, Barbhuiya MA, Stein S, Kunkel H, Garc\u00eda P, Bizama C, Riquelme I, Espinoza JA, Kurtz SE, Tyner JW, Calderon JF, Corval\u00e1n AH, Grez M, Pandey A, Leal-Rojas P, Roa JC. Oncotarget. 2017. doi: 10.18632\/oncotarget.15410.\r\n\r\nRegulation of Connexins Expression Levels by MicroRNAs, an Update. Calder\u00f3n JF, Retamal MA. Front Physiol. 2016;7:558.\r\n\r\nElevated expression levels of lysyl oxidases protect against aortic aneurysm progression in Marfan syndrome. Busnadiego O, del Blanco D, Gonzalez-Santamar\u00eda J, Habashi J, Calder\u00f3n JF, et al. Journal of Molecular and Cellular Cardiology. 2015.\r\n\r\nCurrent Controversies in Diagnosis and Management of Cleft Palate and Velopharyngeal Insufficiency. Ysunza PA, Repetto GM, Pamplona MC, Calder\u00f3n JF, et al. BioMed Research International. 2015; Article ID 196240.\r\n\r\nAngiotensin II-dependent TGF-\u03b2 signaling contributes to Loeys-Dietz syndrome vascular pathogenesis. Gallo EM, Loch DC, Habashi JP, Calderon JF, et al. J Clin Invest. 2014;124(1):448-60.\r\n\r\nGluA3-deficiency in mice is associated with increased social and aggressive behavior and elevated dopamine in striatum. Adamczyk A, Mejias R, Takamiya K, Yocum J, Krasnova I, Calder\u00f3n JF, Cadet JL, Huganir R, Pletnikov M, Wang T. Behavioural Brain Research. 2012;229(1):265-72.\r\n\r\nVEGFA polymorphisms and cardiovascular anomalies in 22q11 microdeletion syndrome. Calder\u00f3n JF, Puga AR, Guzm\u00e1n ML, Astete CP, et al. Biol Res. 2009;42(4):461-8.\r\n\r\nClinical features of chromosome 22q11.2 microdeletion syndrome in 208 Chilean patients. Repetto GM, Guzm\u00e1n ML, Puga A, Calder\u00f3n JF, et al. Clin Genet. 2009;76(5):465-70.\r\n\r\nReconocimientos\r\n\r\nASHG\/Charles J. Epstein Trainee Award for Excellence in Human Genetics Research. American Society of Human Genetics, Boston, MA.\r\n\r\nFulbright \u2013 CONICYT Predoctoral Training Scholarship. 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